| PT FAT LOSS INTENSIVE:
EXERCISE AND OBESITY |
FILEX 2001 Fitness and
Healthy Lifestyle Convention Darling Harbour Sydney 3 June 2001
Robert J Parker, MEd,
FACHPER, PhD Research Fellow, Science Faculty, University of Sydney
Head, Children's Hospital Institute of Sports Medicine The Children's
Hospital at Westmead
There has been a steady
increase in the proportion of overweight and obese adult Australians
over the past 15 years. Trend data from the National Heart Foundation
Risk Factor Prevalence Survey (1989) and the National Nutritional Survey
(1995), show that the proportion of overweight and obese women aged
between 25 and 64 years has increased from 27% in 1980 to 43% in 1995.
Similarly the proportion of overweight or obese men rose from 48% to
63% over this period. The proportion of obese men has increased from
8% in 1980 to 18% in 1995 and the proportion of obese women has increased
from 7% to 16% On average, women in 1995 weighed 4.8 kg more than their
counterparts in 1980.
Comparisons of statistics
between the 1985 ACHPER survey and more recent data highlight an increasing
prevalence of obesity among young populations (Wilcken et al., 1996,
Dolman et al. 1998, Wake 1999). Booth et al have recently made a comparison
of three recent cross-sectional Australian surveys (the National Nutrition
Survey; the Health of Young Victorians survey; and the NSW Schools survey).
Using the new Body Mass Index (BMI) for age- international reference
values, the authors found that approximately 2022% of Australian children
and adolescents are overweight or obese and the trends show that this
rate is increasing. While agreement on the criteria to define obesity
has been problematic (Rowland, 1990), no matter which criteria are applied,
the incidence of children in Australia who can be classified as overweight,
is a major concern.
Several theories exist
to help explain the aetiology of obesity including genetics (Bouchard,
1994; Caterson 1998; Caro et al., 1996) and birth weight (Rona et al.,
1996). Baur (1997) has suggested that while the genetic predisposition
to become overweight in the paediatric population has probably not altered
over the years, the environments in which these genes can fully express
their potential (in Westernised society) has changed markedly. There
are a number of environmental factors, which have been linked to the
prevalence of obesity both in childhood and adults. These include a
decline in the level of energy expenditure and dietary fat intake imbalances.
Effective weight loss strategies
in the adult population would appear to be complex. There is sufficient
data to indicate that restrictive diet interventions in the adult market
can be efficacious in the reduction of body weight. This method however
appears to show a greater proportion of fat-free mass loss relative
to fat mass loss and often results in a decrease in metabolic rate with
a concomitant weight gain with re-feeding. There is also sufficient
evidence to show that exercise alone is not an effective method for
achieving fat loss when compared to the effects of dietary interventions
(Ross et al 2000). Specific exercise modalities have been shown to hold
fat-free mass and either attenuate or increase metabolic rate to pre-weight
loss levels.
Recent attention has focussed
on the importance of exercise intensity on fat loss. It is now clear
that a lower level of aerobic activity (ie., with lower intensity levels)
than that recommended by ACSM (1998) may reduce the risk for certain
chronic degenerative and cardiovascular diseases and improve metabolic
fitness but may not be of sufficient quantity or quality to improve
V02maX (or fitness).
Exercise for health and
fitness is therefore best understood in the context of a dose-continuum.
That is, there is a dose-response to exercise by which health benefits
are gained through varying quantities of exercise ranging from 7002000
plus kilocalories of effort per week. Training volume, rather than training
intensity, may therefore be a more important factor when some health-related
fitness outcomes are desired. The ACSM (1998) guidelines would, therefore,
appear to be aimed at the middle-to-higher end of the exercise continuum.
Aerobic exercise prescription
for fat loss has been questioned recently in the context of this dose-response
continuum. In the unfit, fat individual, where the fitness level is
very poor, use of the ACSM guidelines would appear to be inappropriate,
because the training intensity would be too severe causing a high level
of discomfort during exercise (largely due to a lower LT) and posing
an increased risk of injury.
There is strong evidence
however to show that even higher intensity exercise (e.g. - 85% HRmax)
in non-obese subjects has been shown to provide better fat loss results
than training at a lower intensity level (Tremblay, 1994). High intensity
aerobic exercise has also shown to increase exercise and resting energy
expenditure separate from any change in muscle mass (Hunter et al 1998).
High intensity exercise has resulted in greater fat loss despite less
total energy expenditure when compared to a higher level of energy expenditure
achieved in a lower intensity exercise session (Tremblay, 1994). High
intensity exercise has also been shown to reduce the post-exercise energy
intake compensation (Tremblay, 1999). Fat has been demonstrated to be
the dominate substrate used during high-intensity exercise and during
the brief post-exercise period of "pay-back" when compared to low intensity
exercise (Kriketos et al, 2000). On the other hand, when total exercise-induced
energy deficit is the same, intensity of exercise training has been
reported to have no influence on the size of fat loss in men or women
(Ballor, 1990).
These high-intensity induced
benefits, however, should be weighed against the potential risk of injury
and level of exercise discomfort, and hence, exercise compliance over
time. It is therefore recommended that in the unfit, obese market, low
intensity exercise (i.e. 50-60% of HRmax) should be performed on a daily
basis for longer duration (e.g. -- 60 minutes) with a view of expending
as much energy as possible (ACSM, 1998). This recommendation would account
for an energy expenditure of around 400kcal in a realistic program aiming
for a total energy deficit of 700-800kcal per day. If total body mass
and fat mass are not important considerations in an exercise program,
shorter duration, higher intensity interval-exercise programs are recommended
for improvements in V02maX for healthy individuals at low risk to injury.
Key issues to be discussed
in this session will include: the appropriate intensity of exercise
needed for fat loss (or weight stability); the impact of exercise intensity
on total body weight and fat weight loss; the importance of aerobic
exercise and resistance training on energy expenditure, both during
and after exercise, the role of exercise in the maintenance of fat-free
mass; and the impact of aerobic exercise and resistance training on
substrate utilisation and metabolic rate.
Selected References:
American College of Sports
Medicine (1998) The recommended quantity and quality of exercise for
developing and maintaining card iorespiratory and muscular fitness and
flexibility in healthy adults. Position Statement. Med Sci Sports Exerc
30 (6): 975 - 991.
Baur, L.A. (1997) Obesity
in childhood - consequences, aetiology and management. (Proceedings)
Child Nutrition Today, Health Tomorrow. HINS National Nutrition Symposium,
Melbourne, May. pp 36-45
Bailor, et al., (1990)
Exercise intensity does not affect the composition of dietand exercise-induced
body mass loss Am J. Clin Nutr. 51: 142-146.
Bouchard, C. (1994) Genetics
of obesity: Overview and research directions. In: Bouchard, C. (Ed)
The Genetics of Obesity CRC Press: Boce Raton, FL, pp223-233.
Caro, J.F., Sinha, M.K.,
Kolaczynski, J.W., Zhand, P.L. & Considine, R.V. (1996) Leptin:
the tale of an obesity gene. Diabetes, 45: 14551462
Dolman J, Olds T, Norton
K, and Stuart D. Trends in the health-related fitness of Australian
children (Abstract) Australian Conference of Science and Medicine in
Sport, Adelaide, 1998 (p100).
Feigenbaum, M. and Pollock,
M. (1997) Strength training: rationale for current guidelines for adult
fitness programs Physician and Sports Med. 25: 4464.
Hunter, G., Weinsier, R.,
Bamman, M. and Larson, D. (1998) A role for high intensity exercise
on energy balance and weight control. International Journal of Obesity
22: 489-493.
Kriketos, A. et al., (2000)
Effects of aerobic fitness on fat oxidation and body fatness Med Sci
Sports Exerc. 32 (4): 804-811.
NH&MRC (1997) Acting
on Australia's weight: a strategic plan for the prevention of overweight
and obesity. National Health and Medical Research Council. Australian
Government Printing Service; Canberra.
Ross, R., Jansssen, 1.
and Tremblay, A. (2000) Obesity reduction through lifestyle modification.
Canadian Journal of Applied Physiology 25(1): 1-18
Rowland,
T.W. (1990). Exercise and Children's Health. Human Kinetics, Champaign,
II, USA.
Sallis, J.F. (1995) A behavioural
perspective on children's obesity. In: L. Cheung and J.B. Richmond (eds)
Child Health, Nutrition and Physical Activity. Human Kinetics, Champaign,
IL, pp.125-138.
Tremblay, a. Doucet, E.
and Imbeault, P. (1999) Physical activity and weight maintenance. International
Journal of Obesity 23, Suppl 3: S50-S54.
Tremblay, A. Simoneau,
J. and Bouchard, C. (1994) Impact of exercise intensity on boy fatness
and skeletal muscle metabolism. Metabolism 43: 814-818.
Wake, M. What's happening
to Australian Children? (1999) Australasian Society of the Study of
Obesity. Proceedings from the Eight Annual Scientific Meeting, Sydney,
Australia, p 55
Wilcken, D.E.L., Lynch,
J.F., Marshall, M.D., Scott, R.L. & Wang, X.L. (1996) Relevance
of body weight to apo-lipoprotein levels in Australian children. Medical
Journal of Australia. 164: 22-25.
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